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 1-10    11-20   21-24 
1.
Инвентарный номер: нет.
   
   S 98

   
    Systemic inflammation and liver damage in HIV/hepatitis C virus coinfection / K. V. Shmagel, E. V. Saidakova, N. G. Shmagel [et al.] // HIV Medicine. - 2016. - Vol. 17, № 8. - P581-589. - Bibliogr. : p. 7-9 (40 ref.)
ББК 61
Рубрики: ЗДРАВООХРАНЕНИЕ. МЕДИЦИНСКИЕ НАУКИ
Кл.слова (ненормированные):
ANTIGENS -- HEPATITIS C -- HIV INFECTION

\\\\expert2\\NBO\\HIV Medicine\\2016. 1-9.pdf
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2.
Инвентарный номер: нет.
   
   C 36

   
    CD8.sup.+ T cell expansion in HIV/HCV coinfection is associated with systemic inflammation / E. V. Saidakova, K. V. Shmagel, L. B. Korolevskaya [et al.] // Doklady Biological Sciences. - 2017. - Vol. 474, № 1. - P126-128
ББК 001.2
Рубрики: НАУКА. НАУКОВЕДЕНИЕ
Кл.слова (ненормированные):
HEPATITIS C -- RISK FACTORS -- COMORBIDITY -- RISK FACTORS -- T CELLS -- HEPATITIS C VIRUS
Аннотация: High prevalence of non-AIDS-defining illnesses in treated HIV-infected patients is associated with increased peripheral CD8.sup.+ T cell counts. Hepatitis C virus (HCV) coinfection is an additional risk factor for the development of non-AIDS events. We found that, in HIV/HCV coinfection, the increased proportion of CD8.sup.+ T lymphocytes is due to the effector memory and terminal effector T cells gain. Moreover, in these patients, the accumulation of highly differentiated forms of CD8.sup.+ T lymphocytes was associated with increased concentrations of inflammatory indices.

\\\\Expert2\\NBO\\Doklady Biological Sciences\\2017 v. 474 p. 126-128.pdf
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3.
Инвентарный номер: нет.
   
   I-55

   
    Immunopathophysiology of systemic inflammation / V. A. Chereshnev, E. Gusev , N. V. Zotova, M. V. Chereshneva // 13 th World Congress on Inflammation : program and abstract book. - London, 2017. - P52-53
ББК 54
Рубрики: ЗДРАВООХРАНЕНИЕ. МЕДИЦИНСКИЕ НАУКИ

\\\\Expert2\\NBO\\статьи из сборников\\13 th WCI 2017 Abstract Book С.52-53.pdf
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4.
Инвентарный номер: нет.
   
   Ч-46

   
    Acute and chronic systemic inflammation: features and differences in the pathogenesis, and integral criteria for verification and differentiation / N. Zotova, Y. Zhuravleva, V. Chereshnev, E. Gusev // International Journal of Molecular Sciences. - 2023. - Vol. 24, № 2. - Ст. 1144
ББК Ч2
Рубрики: ЗДРАВООХРАНЕНИЕ. МЕДИЦИНСКИЕ НАУКИ
Кл.слова (ненормированные):
SYSTEMIC INFLAMMATION -- INFLAMMATORY BIOMARKERS -- SEPSIS

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5.
Инвентарный номер: нет.
   
   L 83

   
    Liver damage and inflammation in HIV/hepatitis C virus coinfection [Text] / K. V. Shmagel, E. V. Saidakova, N. G. Shmagel [et al.] // Глобальное партнерство в борьбе с ВИЧ/СПИДом. Ценна каждая жизнь : тезисы пятой конференции по ВИЧ/СПИДу в Восточной Европе и Центральной Азии. - Москва, 2016. - P278-279
ББК 61
Рубрики: ЗДРАВООХРАНЕНИЕ. МЕДИЦИНСКИЕ НАУКИ
Кл.слова (ненормированные):
HIV/HEPATITIS -- LIVER DAMAGE

\\\\Expert2\\NBO\\Электрон. библиотека (Отеч.периодика)\\Черешнев В. А\\Тезисы 5 конференции по СПИДУ 2016-Abstracts P. 278-279.pdf
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6.
Инвентарный номер: нет.
   
   Z 86

    Zotova, N. V.
    Systemic inflammation: methodological approaches to identification of the common pathological process / N. V. Zotova, V. A. Chereshnev, E. Yu. Gusev // PLOS One. - 2016. - Vol. 11, № 5. - P. e0155138
ББК 61
Рубрики: ЗДРАВООХРАНЕНИЕ. МЕДИЦИНСКИЕ НАУКИ
Кл.слова (ненормированные):
MULTI-SYNDROME -- PATHOLOGICAL PROCESS
Аннотация: We defined Systemic inflammation (SI) as a “typical, multi-syndrome, phase-specific pathological process, developing from systemic damage and characterized by the total inflammatory reactivity of endotheliocytes, plasma and blood cell factors, connective tissue and, at the final stage, by microcirculatory disorders in vital organs and tissues.” The goal of the work: to determine methodological approaches and particular methodical solutions for the problem of identification of SI as a common pathological process. SI can be defined by the presence in plasma of systemic proinflammatory cell stress products—cytokines and other inflammatory mediators, and also by the complexity of other processes signs. We have developed 2 scales: 1) The Reactivity Level scale (RL)–from 0 to 5 points: 0-normal level; RL-5 confirms systemic nature of inflammatory mediator release, and RL- 2–4 defines different degrees of event probability. 2) The SI scale, considering additional criteria along with RL, addresses more integral criteria of SI: the presence of ≥ 5 points according to the SI scale proves the high probability of SI developing. To calculate the RL scale, concentrations of 4 cytokines (IL-6, IL-8, IL-10, TNF-α) and C-reactive protein in plasma were examined. Additional criteria of the SI scale were the following: D-dimers>500ng/ml, cortisol>1380 or <100nmol/l, troponin I≥0.2ng/ml and/or myoglobin≥800ng/ml. 422 patients were included in the study with different septic (n-207) and aseptic (n-215) pathologies. In 190 cases (of 422) there were signs of SI (lethality 38.4%, n-73). In only 5 of 78 cases, lethality was not confirmed by the presence of SI. SI was registered in 100% of cases with septic shock (n-31). There were not significant differences between AU-ROC of CR, SI scale and SOFA to predict death in patients with sepsis and trauma.

\\\\expert2\\NBO\\PLoS ONE\\2016, v.11, p. e0155138pdf.pdf
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7.
Инвентарный номер: нет.
   

   
    SARS-Cov-2-specific immune response and the pathogenesis of Covid-19 / E. Gusev, A. Sarapultsev, L. Solomatina, V. Chereshnev // International journal of molecular sciences. - 2022. - Vol. 23, № 3. - Ст. 1716
Рубрики: ЗДРАВООХРАНЕНИЕ. МЕДИЦИНСКИЕ НАУКИ
Кл.слова (ненормированные):
КОРОНАВИРУС -- COVID 19 -- AUTOIMMUNITY -- ADAPTIVE IMMUNITY -- CELLULAR STRESS -- SARS-CoV-2;
Аннотация: The review aims to consolidate research findings on the molecular mechanisms and virulence and pathogenicity characteristics of coronavirus disease (COVID-19) causative agent, severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), and their relevance to four typical stages in the development of acute viral infection. These four stages are invasion; primary blockade of antiviral innate immunity; engagement of the virus’s protection mechanisms against the factors of adaptive immunity; and acute, long-term complications of COVID-19. The invasion stage entails the recognition of the spike protein (S) of SARS-CoV-2 target cell receptors, namely, the main receptor (angiotensin-converting enzyme 2, ACE2), its coreceptors, and potential alternative receptors. The presence of a diverse repertoire of receptors allows SARS-CoV-2 to infect various types of cells, including those not expressing ACE2. During the second stage, the majority of the polyfunctional structural, non-structural, and extra proteins SARS-CoV-2 synthesizes in infected cells are involved in the primary blockage of antiviral innate immunity. A high degree of redundancy and systemic action characterizing these pathogenic factors allows SARS-CoV-2 to overcome antiviral mechanisms at the initial stages of invasion. The third stage includes passive and active protection of the virus from factors of adaptive immunity, overcoming of the barrier function at the focus of inflammation, and generalization of SARS-CoV-2 in the body. The fourth stage is associated with the deployment of variants of acute and long-term complications of COVID-19. SARS-CoV-2’s ability to induce autoimmune and autoinflammatory pathways of tissue invasion and development of both immunosuppressive and hyperergic mechanisms of systemic inflammation is critical at this stage of infection.

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8.
Инвентарный номер: нет.
   

   
    In HIV-infected immunological non-responders, hepatitis C virus eradication contributes to incomplete normalization of systemic inflammation, but does not lead to rapid CD4+ T-cell count recovery / E. V. Saidakova, L. B. Korolevskaya, N. G. Shmagel [et al.] // Doklady Biochemistry and Biophysics. - 2023. - Vol. 512. - P274–278
Рубрики: ЗДРАВООХРАНЕНИЕ. МЕДИЦИНСКИЕ НАУКИ
Кл.слова (ненормированные):
HIV INFECTIONS -- HEPATITIS C -- IMMUNOLOGICAL NON-RESPONSE
Аннотация: In HIV-positive individuals taking antiretroviral therapy, coinfection with hepatitis C virus (HCV) increases systemic inflammation, which interferes with the CD4+ T-cells regeneration. This study evaluated the effect of HCV eradication on systemic inflammation and CD4+ T-cell regeneration in patients who gave poor response to antiretroviral therapy, the so-called “immunological non-responders” (INRs). HIV-infected patients who received a course of direct-acting antivirals for treating hepatitis C were examined. The control groups included HIV/HCV-coinfected INRs and relatively healthy volunteers. It was established for the first time that HCV eradication is not accompanied by a complete suppression of systemic inflammation, but improves the T-cell pool composition: in INRs, the blood CD4+/CD8+ T-lymphocyte ratio increases and approaches those of healthy individuals. Apparently, in INRs treated for hepatitis C, the immune system recovery takes time and may be incomplete.

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9.
Инвентарный номер: нет.
   

   
    Role of proinflammatory cytokines in hashimoto's thyroiditis associated with psychiatric disorders / P. A. Sobolevskaya, A. N. Gvozdeckii, I. V. Kudryavtsev [и др.] // Medical Immunology. - 2023. - Vol. 25, № 5. - С. 1247-1252
Рубрики: ЗДРАВООХРАНЕНИЕ. МЕДИЦИНСКИЕ НАУКИ
Аннотация: Mental disorders often accompany autoimmune diseases, for example, since 1949 it has been known about “myxedematous madness”, a psychosis caused by hypothyroidism. The most common cause of hypothyroidism is Hashimoto's autoimmune thyroiditis. It is also known about another neuropsychiatric disorder associated with autoimmune thyroiditis, Hashimoto's encephalopathy. It is a severe dysfunction of the central nervous system, the pathogenesis of which is not associated with hormonal disorders. Cytokines are regulators and participants of inflammation, including autoimmune. Certainly, when we are talking about high concentrations cytokines, we mean systemic inflammation. The minimal or mediocre fluctuations in cytokines within the ranges that are characteristic of healthy status or normergic acute phase response in disease cannot be interpreted from the point of view of binary endocrinological logic. In the CNS, cytokines are able to influence on the neuroendocrine control of systemically regulated functions. It is also important that glial cells (astroglia, microglia) are capable of producing a number of cytokines and can affect neurons and develop behavioral changes. In addition, the ability of a number of cytokines outside the CNS itself to act on vagal afferents and through them to convey information to the CNS, affecting its state and functions, has been proven. It is reasonable to assume that minimal fluctuations in cytokine levels may also affect the state and function of the CNS. The aim of the study was to investigate the levels of cytokines in patients with thyroiditis; in patients with thyroiditis associated with mental disorders; in a group of healthy individuals; and evaluate the effect of cytokine levels on clinical manifestations. In the group of patients with thyroiditis and mental disorders, the levels of CCL20/MIP3α, IL-13, IL-2, IL-27, IL-5 were significantly higher than in other groups. At the same time, no positive correlation was found between the clinical manifestations of mental disorders and the levels of cytokines. A positive correlation was found between the levels of some cytokines and free triiodothyronine, as well as the level of antithyroid antibodies. Mental disorders associated with autoimmune thyroiditis may be associated with changes in the cytokine profile and result from neuroinflammation.

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10.
Инвентарный номер: нет.
   
   H 65

   
    High level of sistemic inflammation in HIV/hepatitis C coinfection is linked to hepatocellular injury / E. V. Saidakova, K. V. Shmagel, L. B. Korolevskaya [et al.] // Tokyo – Moscow International Medical Forum : program & abstract book. - Moscow, 2018. - P73
ББК 53
Рубрики: ЗДРАВООХРАНЕНИЕ. МЕДИЦИНСКИЕ НАУКИ

\\\\Expert2\\NBO\\Электрон. библиотека (Отеч.периодика)\\Черешнев В. А\\Tokyo – Moscow International Medical Forum 2018 P.73.pdf
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